Even before I found this article http://www.rense.com/general32/beef.htm I thought about the correlations between the tissue where scientists test for the prion that causes Mad Cow disease (Bovine Spongeform Encephilapathy the human variation being CJD or Cruetzfeld Jacobs Disease) and the missing pieces taken from cows starting as far back as the mid sixties.
In the mid sixties was also when the deer/elk variant of BSE called Chronic Wasting disease was released into the deer and elk populations of North America. The release was accidental and hardly talked about but was caused by an experimental elk herd given the same types of feed that gave cows the disease in the first place (meat and bone meal from infected cows recycled and fed back to cows or in this case elk).
Since that time the cattle mutilations have become more frequent and brazen, with the ever familiar black helicopters buzzing ranches and farms, with no explanation why. Many people are beginning to think that the mad cow disease is actually rampant in the system due to the unregulated use of so called downer cows which are never tested before they enter the system.
In the last couple of years in the media it was reported one case of BSE was found in the US, and many cows were slaughtered, I believe this was all just hype. Once this prion is in the system its impossible to get rid of, its not a virus, it cannot be killed and its been burned in temperatures past two thousand degrees and remains unchanged. Cases of the human form have been found to have been transmitted by surgical tools due to the fact that regular sterilization procedures do not eraticate the prion.
Will all of this the cases of "Altzhiemers" disease have skyrocketed, with the disease nearly identical in its symptoms, and only discernable way to tell the difference is through post mortem brain slides. I truly believe the government knows about BSE being rampant in the beef industry and has kept its own eye on the situation by taking cattle for testing and keeping the whole thing secret...what do you all think???

here are some addtional links I found that were of interest:
http://www.shout.net/~bigred/MadCows.htm this one has some close up pics so they may be disturbing to some
http://users.rcn.com/jkimball.ma.ultranet/...s/P/Prions.html
this one is all about prion diseases in general but gives info on the human form spread by surgical instruments

By far, this is the most convincing, sensible explanation for cattle mutliations that ive read. Alternative explanations are far fetched (to say the least) one bright and shining example

Thank you darkmoonlady for researching this and shedding some light on an otherwise unexplained phenomenon.

This is very interesting.

Well that does gie you something to actually think about.

yea preons are funny little critters --er-if there alive-- there closer to some kind of protien/dna baised toxin that self replicates . the stuff is soo tough that you can't even burn the stuff and get rid of it.

if i didn't know better i would suspect it is some kind of nanu-botic biological agent.

but i'm too far out of my field to hazzard a WAG.

An article about cattle mutilation that this farmer seems to have a lot of experience with. I haven't heard this info. before. This guy seems to know what he is talking about.

hit latest 100 messages: Cattle Mutes...Fern Belzil...Sept. 07/04

here

Here's another take.

Cattle Mutes_King....Kyle King....Sept.09/04

hit latest 100 messages

here

After a certain length of time you will have to do a search at the site. Under latest 100 messages is a google search. Just search for the article above.

Something to think about endeed.

What was it that John Titor said about mad cow disease? LOL
*thankful to be a vegetarian* and thanks for sharing your finds in this subject

Here is a bit of an update, these two articles on MSNBC news one about the disease in deer, which in the article they try to say that its not the same as mad cow, and is caused from something different, and then say it is the same, its all that run around keeping the public from understanding that mad cow is in the US and its all through the system. Mule deer are often seen grazing in areas with cattle on public lands, and ranches.
http://msnbc.msn.com/id/5463499/

and her is an acticle on a woman who died of mad cow and may have infected other patients with it

http://www.msnbc.msn.com/id/5772569/


In the deer article it states that no case of chronic wasting disease has been found to have become CJD (creutzfeld Jacob disease the human varient) in any people. I believe this is untrue. I have seen television news articles about a hunter dying of it. I will try to find this info and post it as an addition to this one.

Yet another suspected case of mad cow disease in Washington state. I think the time is finally coming when it is going to come out that mad cow disease is happening just as often here as in Europe.


http://www.cnn.com/2004/HEALTH/conditions/...w.ap/index.html

why would the government do this so secretly? i'll admit i did not read the above links but, why would they hide the fact they were researching the mad cow disease? and why would they to go to the lengths of mutilating cows and not just purchasing there own or purchasing them from the farmers them selves?

Coz everybody loves a good conspiracy, that's why.

so what is it in the virus that makes the cow turn inside out? im not making the connection?

in this case what if govt accidentally find virus in their mutilated materials? will they keep silent about the whereabout of the potential
dangers?

I read some were that the prion could be killed using steam heat up tp 10,000 degress.I can't remember were I read that thow..

I'm afraid this is not the reason for the cattle mutilations. We must remember that cattle mutilations have taken place in almost every country in the world, not just the U.S., and Seeking has a good point. We're talking about a government with enough money and power to buy the whole world, and they can't afford to purchase some little ol' cows? I find that hard to believe. The culprit is -->

Me too.



I was hoping we could get further thru our list of possible culprits before we landed on aliens, we didn't even try ghosts, or the grinch. How about fairies?

Then I'm afraid you are on the wrong website.

There are tons of deer in my area (so many that is becoming a problem) and some of these deer are known to be infected by this "wasting disease". It is a common subject around here. But my familly use to raise cattle untill just a few years ago and none ever came down with wasting disease nor mad cow disease (I can't recall any reports from other farmers around here either). Also, there have been no cattle mutilations, or strange black helicoptors were I live.

But the question is, why would they not release it to the public? Why keep it a secret? I mean if it is to test for mad cow disease...

The only thing I can think of is so animal rights groups don't protest it. o-o;

I just saw this interesting article here....

http://www.pamrotella.com/health/bovinebacteria.html

http://drbroxmeyer.netfirms.com/

The bacterial model of Mad Cow Disease
[Posted 26 September 2004, last updated 19 November 2004]

The mainstream media has embraced the prion theory of Mad Cow Disease since BSE ( Bovine Spongiform Encephalopathy, known as Mad Cow Disease) became an epidemic in the 1990s. Other than a few alternative news sources, the press largely ignores other theories of the disease. The bacterial model of Mad Cow Disease is a competing theory with extensive supporting scientific evidence. In his article Is Mad Cow Disease caused by a bacteria?, Lawrence Broxmeyer, M.D., presents documentation supporting the Bovine Tuberculosis model of Mad Cow Disease.

Broxmeyer is a researcher who has concentrated much of his time on Tuberculosis (TB), and is familiar with how TB can cause symptoms like those found in the Spongiform Encephalopathies. His full article Is mad cow disease caused by a bacteria? (.pdf version) can be found by clicking here.* The text version (does not include epidemic maps) can be found by clicking here.

Bovine TB has a long history in Great Britain, with symptoms often similar to those of Mad Cow Disease. The deformed "prions" implicated in other research may be another symptom of the disease and not the cause, according to Broxmeyer, and "many animals that die of spongiform TSE's never show evidence of misfolded proteins." Broxmeyer says the infectious capability of a protein without genetic material is unlikely, and "Prions" were found to be proteins occurring naturally in normal tissues, although changes were sometimes noticed in the diseased animals. However those prion changes wouldn't explain damage to healthy tissues, and a causative agent is needed to explain prion "misfolding" at the outset. Moreover, prions have not been proven as necessary to cause the disease, and an increased level of prions in laboratory experiments has not been shown to increase the likelihood of infection. What researchers are calling prions, according to Broxmeyer, could in fact be "amyloids," a type of "deposition that took place due in the course of chronic inflammatory disease, mainly tuberculosis, the usual precipitating cause." Britain's historical battle with Bovine TB is documented by Broxmeyer, with the disease often appearing in the best stables, and creating a slight risk of infection to humans consuming the flesh of diseased animals. Broxmeyer provides maps to show that the concentration of bovine TB in the southwestern area matches the outbreak of mad cow disease. He claims that the link between eating beef diseased with Bovine TB and the human disease is well established.

Broxmeyer, a doctor who has treated TB in patients and studied it extensively, cites symptoms of TB which match the encephalopathy and neurological damage seen in Mad Cow Disease (BSE), Scrapie in sheep, and CJD in humans. He explains that Tuberculosis often assumes "L-forms," or cell-wall deficient forms, which are hard for researchers to detect using standard methods, and evade the animal's immune system. (This is also related to the bacterium's pleomorphic nature.) Mad Cow tissue was shown to be infectious in experimentation even without "prions" present, which could indicate that an agent like L-forms are at work. Bovine TB can also cause "downer cows" and both meningitis and encephalitis in cattle and humans.

Current mad cow diagnosis lies solely in the detection of late appearing "prions", an acronym for hypothesized, gene-less, misfolded proteins, somehow claimed to cause the disease. Yet laboratory preparations of prions contain other things, which could include unidentified bacteria or viruses. Furthermore, the rigors of prion purification alone, might, in and of themselves, have killed the causative virus or bacteria. Therefore, even if samples appear to infect animals, it is impossible to prove that prions are causative.
Lawrence Broxmeyer, M.D., Is mad cow disease caused by a bacteria?

I asked Dr. Broxmeyer about Mark Purdey's theory of Mad Cow Disease, the organophosphate/manganese poisoning model. Purdey's theory of an organophosphate-Mad Cow link started with the very compelling observation that Purdey's organic herd, untreated with organophosphate pesticides, did not contract Mad Cow Disease, whereas a non-organic herd of his own which had been treated and his neighbors' treated herds all contracted Mad Cow Disease. Also, the Mad Cow epidemic immediately followed the British government's mandatory treatment of cattle with organophosphate pesticides in order to "eradicate" the warble fly parasite. This was a very strong observation, and usually such observations are eventually proven correct by science.

Although Broxmeyer doesn't think that the actual cause of Mad Cow goes beyond Bovine tuberculosis, and that blaming disease on agents such as organophosphates was a traditional flaw even in the history of discovering the real cause for tuberculosis, he admitted that there could be an indirect linkage with not only organophosphates but a host of other chemical and physical irritants. According to Broxmeyer, "Mankiewicz and Livingston's colleague Alexander-Jackson (1965) established that bacteriophages (also called phages), the viruses which live inside pathogens such as bovine and human tuberculosis could in and of themselves cause cytopathogenic change, even pre-malignancy in otherwise normal healthy mammalian tissue. Lwoff (1962) and early phage masters showed how the phages inside Mankiewicz's mycobacteria could be activated by a host of chemical and other agents, some pesticide-like, including organophosphates. Organophosphates therefore could be one of many irritants causing Mad Cow clinically in bovine tuberculosis infected cattle by causing them to activate the phage viruses within this disease. And since Nelson and Pickett (1951) showed that it was attack by these very same phage viruses which seemed to cause the majority of Klieneberger's cell-wall-deficient (pleomorphic) forms thru breech of the cell wall, organophosphates would indirectly cause them to propagate and the underlying disease in Mad Cow, central nervous system bovine tuberculosis would become aggravated."

Broxmeyer however, because of the intricacies of such an organophosphate relationship and the fact that he felt Bovine TB itself, and not organophosphates, causes Mad Cow, chose not to cover that particular issue in his article. On the other hand, he draws freely from the "concept of Klieneberger and Livingston's viral (pleomorphic) forms of Bovine tuberculosis" to explain why some investigators might interpret that a virus is behind Mad Cow. "Of all the pathogens," he relates, "the preferred form of any tuberculosis or the mycobacteria is the cell-wall-deficient or pleomorphic state, a strategy they [the bacteria] have devised to create dormant forms which both go beneath the body's radar of detection and mean guaranteed survival." Obviously, with pleomorphism a forbidden yet well-known secret in the scientific community, little research would be funded or published to back such claims. Broxmeyer is one of the few researchers brave enough to cite Virginia Livingston as it is. (See my article on the Genetic Fad for a brief explanation of pleomorphism.)

There is no known disease which better fits into what is occurring in Mad Cow and the spongiform encephalopathies than bovine tuberculosis and its blood-brain barrier penetrating, virus-like, cell-wall-deficient forms.
Lawrence Broxmeyer, M.D., Is mad cow disease caused by a bacteria?

http://drbroxmeyer.netfirms.com/



http://www.pamrotella.com/

ok explain to me then, why they do it undercover without the farmers permission, and dont just breed cows them selfs and do the tests..

Might be a stupid question but even if they decide not to breed their own and want statistically random test subjects to check on disease prevelence why don't they just take the whole cow back to the lab rather than leave mutilated carcasses for nutjobs to speculate about. Surely a stolen or missing cow is simply a stolen or missing cow whereas a mutilated one is 'evidence' of supernatural/strange goings on.

Interesting. This is not a new theory. I seem to recal NIDS / bigelows group released a research paper on this.
They connected it to Cronic Wasting Disease and its spread from maryland bio research facilities in the 50's. Incidentally this is the same time period that infected human brains were being sent from cannibal tribes to Maryland for research. Cattle mutilations have steadily moved west since then. Additionally more than one cow has been found with residual anisthetics in the tissue samples.

...an interesting article here....

Copper deficiency!!!!!

http://bse.airtime.co.uk/copper.htm



It is curious that the geographic occurrence of "mad cow" disease (number of cases per 1000 head) is not evenly or randomly distributed in the UK, but has tended throughout the epidemic to be highest in the southern and eastern counties (Wilesmith et al., 1992a). Several counties in this region are known to have widespread copper defiencies in soils and crops (Thornton and Webb, 1980). These crop deficiencies could lead to copper deficiency in ruminants, a fairly well-recognized disease with specific symptoms, in those regions without copper supplements in rations. BSE has tended to have higher occurrence in particular herds, even though there is no definitive evidence that the disease can be transmitted animal-to-animal.Since consumption of MBM presumeably varies from animal to animal, the impact on some animals could be much greater than on others.

"The use of animal protein, which increases nitrogen in the feed, could lead to a deficiency of essential fatty acids in the cell membranes, reducing membrane integrity , and making the animal more susceptible to encephalomalacia (Crawford et al., 1991). The fairly recent increased use of canola seed cake in animal rations in the UK could also contribute to this nutritional imbalance, as canola has a high sulfur content and can accumulate certain toxic metals from soils in the seed. Feeds which are high in molybdenum relative to copper are well-known to induce copper deficiency in ruminants (McDowell, 1985), and legume forages or soybean meal can have unacceptably high molybdenum content if grown on non-acid soils with more than 3 ppm molybdenum (McBride, unpublished data). This disease is referred to as molybdenosis, and is generally recognized by obvious symptoms such as changes in hair coat pigmentation.

Based on the number of references that can be found where Cu deficiency has been diagnosed in numerous ruminant species in the wild, as well as captive or in a farming environment, Cu deficiency appears to be common. It has been observed in cattle, moose, red deer, Sika deer, elk , muskoxen and goats (Mackintosh,1998 ; Stafford, 1997; Blakley et al.,1998; Arnhold et al., 1998; Gogan et al., 1989), particularly in cases where wild animals have been captive or confined. Wapiti (elk) may be particularly susceptible to Cu deficiency, and the disease is reported frequently in red deer on farms (Blakley et al., 1992). Confining wild ruminants on farms appears to increase the risk of certain diseases, including copper and other trace element deficiencies (Mackintosh, 1998). Wild ruminants may be able to compensate for soil deficiency of particular micronutrients by obtaining a more varied diet than confined ruminants restricted largely to grass forage (Stafford, 1997). Interestingly, wild ruminants appear to be better adapted to low-Cu diets than most domesticated ruminants, as the necessary level of Cu in the liver tissue of domesticated sheep and cows ( 35 mg/kg dw) is higher than that for deer (10-20 mg/kg) (Arnhold et al., 1998). Domestic goats require even less Cu (8 mg/kg in liver), and it is interesting that the Cu level in the cerebrum is a more reliable indicator of Cu deficiency in goats than that in the liver (Arnhold et al., 1998). Cerebrum Cu concentrations in goats are generally less than 10 mg/kg (dw), levels considered to be marginal or low in sheep and cattle. "

I really dont think these farmers are blaming a unexplained phenomnen when really its just mad cow desease.... it dosent make sense

Ok if you want Whacky....here is what I've found during my googling forays...


http://jtcs.ctsnetjournals.org/cgi/content/abstract/124/1/35

Polymerized bovine hemoglobin solution as a replacement for allogeneic red blood cell transfusion after cardiac surgery: Results of a randomized, double-blind trial..This clinical trial was sponsored by the Biopure Corporation, which provided the polymerized hemoglobin solution used in this clinical trial and provided financial support for the clinical study nurses and additional laboratory analyses required by the clinical protocol.

Received for publication Feb 12, 2001. Revisions requested May 17, 2001; revisions received Oct 30, 2001. Accepted for publication Nov 7, 2001. Address for reprints: Gus J. Vlahakes, MD, Division of Cardiac Surgery, Massachusetts General Hospital, 55 Fruit St-BUL119, Boston, MA 02114-2696 (E-mail: vlahakes.gus@MGH.Harvard.edu).
"tle team in action
The Newcastle team is pioneering stem cell work
UK scientists have applied for permission to create embryos by fusing human DNA with cow eggs.

Researchers from Newcastle University and Kings College, London, have asked the Human Fertilisation and Embryology Authority for a three-year licence.

The hybrid human-bovine embryos would be used for stem cell research and would not be allowed to develop for more than a few days.

But critics say it is unethical and potentially dangerous.

Liberal Democrat MP Dr Evan Harris - a member of the Commons Science and Technology Select Committee - said: "If human benefit can be derived by perfecting therapeutic cloning techniques or from research into subsequently-derived stem cells, then it would actually be immoral to prevent it just because of a 'yuck' factor."

Stem cell research is one of the most promising areas of medical science.


You may begin to undermine the whole distinction between humans animals and humans
Calum MacKellar

Why create chimeras?

Stem cells are the body's master cells and five-day-old embryos are packed with them - each with the potential to turn into any tissue in the body.

It is this ability which scientists want to harness to treat diseases such as Parkinson's Disease, strokes and Alzheimer's Disease.

To do that, they say they need to have access to thousands of embryos for research.

Short supply

The problem is that human eggs for research are in short supply and to obtain them women have to undergo surgery.

That is why scientists want to use cows' eggs as a substitute.

They would insert human DNA into a cow's egg which has had its genetic material removed, and then create an embryo by the same technique that produced Dolly the Sheep.

The resulting embryo would be 99.9% human; the only bovine element would be DNA outside the nucleus of the cell.

It would, though, technically be a chimera - a mixing of two distinct species into one.

The aim would be to extract stem cells from the embryo when it is six days old, before destroying it. "



Then look at what they found in the Red Rains of Kerala!!!!!

http://education.vsnl.com/godfrey/

the particles vary from 4 to 10 microns in size and appear under magnification as red-colored glass beads. Electron microscope work shows them to have “…a fine structure similar to biological cells.”

Kerala's red rain

"And although they look something like unicellular organisms, the particles show no nucleus, although dyes reveal ‘…a layered structure after the dye penetration.’ They’re also quite stable over time, showing no decay or discoloration after storage without preservatives for over four years. No trace of RNA or DNA can be found."



http://iangoddard.net/redrain.htm


..?????altered bovine cells???


"The resulting embryo would be 99.9% human; the only bovine element would be DNA outside the nucleus of the cell."

Weird!!!!

More...on Kerala.....

http://www.neaiga.org/strange_science.htm

RED RAIN is said to contain live DNA

Scientists in Britain say they have confirmed that DNA, the genetic blueprint for life, does exist in the mysterious red rain which fell over the Kerala region of India, in 2001.

The blood-colored rain caused a storm of controversy among the world’s scientists. Many theories have been put forward to explain the strange phenomenon, but the latest results, from studies carried out at Cardiff University in Britain, seem to confirm that the red color does come from living cells, although where they came from remains a mystery.

The strange cells fell as red rain for six weeks, following reports of an explosion in the sky. Indian scientists who first analyzed the rain expected to see grains of dust or sand, perhaps blown from the Sahara by freak winds.

***WATCHMAN COMMENT... There have also been reports all over the USA concerning mysterious BOOMS or EXPLOSIONS being heard that cannot be attributed to sonic booms or other know phenomena...

Instead, they found themselves looking at complex cell-like structures, that have many of the characteristics of living organisms.

They were even more surprised to find the cells could be made to come to life and reproduce, under laboratory conditions.

“If there was an explosion of a small piece of a comet over Kerala, and an explosion was in fact heard just minutes before the first rainfall, those particles would have drifted along a belt of latitude, but when you look at a map of the world, the latitudes west of Kerala run into the Indian Ocean and then into the Sahara.

***WATCHMAN COMMENT... BUT was it a comet? There have been reports of tests releasing spores from planes to see how and where they would spread, supposedly testing a terror attack....

So if it fell all over that area it wouldn’t have been noticed, and in the Sahara there is not much rainfall, so the particles could have drifted a long way away and not be noticed.”

Intensive investigation under high-powered microscopes confirmed the cell-like structures are biological and that they do contain DNA, the blue print of all life forms on Earth.

What’s not yet known is whether it is terrestrial life or alien DNA, but investigators believe they will know soon.

The Cardiff team is now comparing DNA from the red rain with that of all known terrestrial species.

"It’s a long and painstaking study, but if no known DNA from Earth matches, the only remaining possibility would be that it is an alien life form from outer space."

***WATCHMAN COMMENT... There is another possibility! There have been reports that many scientists believe that the world's population MUST be REDUCED! Some say 50% while other say 90% - Believe it! That is TRUE!...

http://www.genengnews.com/news/bnitem.aspx...32&taxid=34
Prion proteins found in commercial milk

In a first-time global breakthrough, a Swiss start-up firm has succeeded in detecting prion proteins in the milk of humans, cows, sheep, and goats. This again raises the question of a "mad cow disease" risk from drinking milk. Tests are underway to verify disease-causing prions in milk.

Prions are known to be causes of neurological conditions such as Mad Cow disease (BSE) and Creutzfeldt-Jakob disease in human beings. The causative agent destroys the central nervous system in humans and animals. It is known that prions can also emerge in body fluids such as blood and be transferred by them. In the past it was difficult to estimate the risk of an infection through blood transfusion or drinking milk, since the concentration of prions in body fluids is very low; nor is there a sensitive method to identify prions. Moreover, the incubation time for infection in human beings can take 10 years or longer.

Prion proteins in milk from the supermarket