What's hair mercury????





http://www.earthtym.net/merc-links.htm


Amalgam "cover up" exposed in Canada (7 K)
http://home.sol.no/~reiersol/canada.htm
(2004 - This page is no longer hosted by ISP, now http://www.sol.no)
March, 1997, from The Bio-Probe Newsletter

"CBC French Network program 'Decouverte' (Discovery) ... which reports on scientific issues which might be covered up ran a 16-minute report on the health risks of amalgam following publication of Health Canada's Position Paper on the issue. ...."

"There followed a public outcry in dental offices throughout Quebec. ..."

"On 23 February, Decouverte aired a second report on amalgam, this time exposing Health Canada's failure to inform Canadians of the possible dangers of this material. Since 1976, four Health Canada reports by medical scientists have warned of these dangers; only in 1996 did Health Canada publish a position statement ...."

"Since 1982, according to Canadian law, all medical devices ... must have pre-market studies done to prove their safety and biocompatability. The president of the Canadian Dental association admitted his predecessors had lobbied the government to exclude dental restorative materials ...."

Dr. Pierre Blais, former risk analyst with Health Canada and author of its 1976 report on amalgam ... showed how the continued use of amalgam was contrary to Health Canada's policy ... concerned over (tiny) amounts (of mercury) in apples and looked the other way when (large) amounts, a million times more, were being directly implanted into a child's mouth." ...

"Dr. Mark Richardson's studies on the relative risks of mercury in amalgams and biphenyl-A in composites were quoted: The average Canadian, with 8 amalgams, was 3.5 times over the daily threshold limit for mercury exposure, whereas the patient with 8 composites was 125 time UNDER the daily threshold limit value for escaping biphenyl-A."

"... the dental patient must be aware and take responsibility for his/her own health." ...


"Anti-amalgam" epidemiological evidence. (26 K)
WAS: http://home.sol.no/~reiersol/evidence.htm
(2004 - This page is no longer hosted)
August 6, 1997

* Parkinson's disease linked to the number of amalgam fillings before onset of the disease.

* Parkininson's disease linked to blood mercury ....

* Heart disease linked to hair mercury and fish consumption ...

* NAG in urine (indicates kidney damage at high levels) increases with increasing number of amalgam fillings ...

* Still births and birth defects linked to mercury in maternal blood and umbilical cord blood ...

* Adolescents with fillings have more anxiety, tension, and other "personality" problems ...

* People with amalgams smoke more than those without (or with few) amalgams. ...

* In patients with tongue cancer and dental fillings only on one side, the cancer is usually on the same side as the fillings. ...

* DMSA (a chelating agent that removes mercury from the body) decreases fatigue in a placebo-controlled study. ...

* Women with amalgams are more depressed, angry and anxious than those without amalgams. ...

* People with amalgams have higher blood pressure, lower heart rate, lower hemoglobin, more chest pains, tachycardia, anemia, and fatigue. ....




German scientists say amalgam reduces fertility. (3 K)
http://home.sol.no/~reiersol/heidel.htm
(2004 - This page is no longer hosted by ISP, now http://www.sol.no)
1997, Oslo, Norway

"A link has been found between mercury and hormone disturbances, immune disturbances, recurrent fungal infections, hair loss and allergies. The differences are large. Allergies and hair loss are 2-3 times as common in the high-mercury group. --- can reduce fertility --- mercury affected hormone productions at low concentrations. ...

My dear, I didn't see anything called hair mercury in the article, but what I think it is refering to, is hair loss, among many other health issues, due to mercury poisoning from canadian dental practices.

* Heart disease linked to hair mercury and fish consumption ...

at first I thought they meant using hair to diagnose toxicity... like they do to test drugs in the system....

That's what they do crystal. They can test the levels of mercury in the hair, just like drugs.

It could also be that people with these traits, more anxious, are less fastitious about tooth care prompting them to have more fillings too! Nonetheless, it probably is the metals that they are putting the teeth. They tell us all the time that we shouldn't eat too many fish because of the merdury but, then they tell us that it's safe to put it directly in out teeth? Some body buy those people a calculator!

Heavy metals tend to permeate the body, including hair. As in most all heavy metal poisoning, hair is tested.
But it is a fact that mercury, along with most other metals (or other chemicals, for that matter), is ubiquitous, just like gold. It is all over the place. Mercury was used frequently in gold acquisition, such as on sluice plates and so on, during the years 1849-1941, and still is used. However, if you go out mining and actually look at the river, you'll find that there are unique properties of mercury. If, for instance, you have a teaspoon of mercury and you drop it in the river or creek, it's gone almost instantly. Mercury finds its way through the overburden within minutes (depending on thickness of overburden) to the bedrock or false bedrock. It doesn't dissolve in the water on its way, either. I've found puddles of mercury on bedrock below 150 year old sluice sites. The dredge sucks up mercury and I hang on to it for later use.
Teeth with amalgam fillings will release small amounts of mercury into the body because of the digestive fluids (acids). The easy way of dealing with heavy metal poisoning is to keep some zinc tablets around, and every now and then take one (dissolved under the tongue). Zinc has been used for decades to counter mercury exposure by chelating the metals and allowing the body to naturally dispose of them.

Thanks for the imformation... I googled it some more and came up with this ....

http://www.nigms.nih.gov/News/Briefs/ZincMercury.htm
Stephen Lippard, Ph.D., of the Massachusetts Institute of Technology in Cambridge may have a new way. He has developed sensors that reveal tiny amounts of zinc and mercury in cells, tissues, or water. When Lippard applied these chemical sensors to biological samples and then shone light on them, molecules of zinc or mercury in the samples lit up. Lippard tested the zinc sensors on brain tissue from rodents with head trauma or seizures and found that the sensors precisely identified zinc in damaged nerve cells. Lippard has also fashioned a sensor that selectively pinpoints even low levels of mercury in water.

The fluorescent chemosensors may offer new tools for imaging metals in the body and for studying the role of these molecules in health and disease. The technology may also prove useful for monitoring environmental quality in water, soil, and elsewhere.



??? Was the zinc in the damaged nerve cells repairing them????



http://www.wanttobewell.com/articles15.htm

Heavy Metals Begone! Eat Cilantro Every Day.
http://curezone.com/forums/fm.asp?i=33657


http://www.chelationtherapyonline.com/articles/p27.htm

http://www.newtreatments.org/

There is some possibility that zinc is an aid to bodily repair.
I say this because there is an old, old remedy that seems to work fairly well: zinc oxide ointment. The stuff seems to help the body repair bruises. Also, I've personally noticed that there is little difference in using triple antibiotic ointment and plain petroleum jelly. Best repair aid though, is pulverized comfrey root. That stuff works wonders. I'm quite certain that most drugs are unnecessary, that if there is some small help given, the body will make repairs in short order.
Anyway, when I was out playing in the gold fields on weekends, using mercury for separation, I always kept zinc tablets with. The reason for this was given me by a mining company, whose equipment was oriented towards gold amalgam (activated mercury and gold particles). The company dissolved base metal hydroxide in water, then used electrodes to insinuate the base metal into mercury metal, the whole of which was quite attractive to gold, even when imbedded in black sands. Their research came from treatments previously used in the medical profession. There was a time when miners who weren't too bright cooked off the mercury in the amalgam using a potato split in half and hollowed out a bit. The amalgam was placed in the hollow, the spud wired shut, and baked over a fire. On opening the spud, the gold button would be where the amalgam had been. Some of the less intelligent ones then ate the cooked spud, incurring mercury poisoning, usually fatal. It doesn't take much mercury to kill a person, especially if ingested, or if it is consistently applied to the skin. Bodily fluids will dissolve small amounts of mercury, then absorb through the pores, eventually causing mild to severe poisoning. One good whiff of mercury vapor is sufficient also. The mining company researched the methodology of treatment, since use of mercury was prevalent in their equipment.

The history of local anesthetic agents.

http://www.doctorspiller.com/local_anesthetics.htm


Surprisingly, the first local anesthetic was Cocaine which was isolated from coca leaves by Albert Niemann in Germany in the 1860s. The very first clinical use of Cocaine was in 1884 by (of all people) Sigmund Freud who used it to wean a patient from morphine addiction. It was Freud and his colleague Karl Kollar who first noticed its anesthetic effect. Kollar first introduced it to clinical ophthalmology as a topical ocular (eye) anesthetic. Also in 1884, Dr. William Stewart Halsted was the first to describe the injection of cocaine into a sensory nerve trunk to create surgical anesthesia. Halsted was an eminent surgeon who had been trained in Britain. He was the first to establish formal surgical training for physicians in America. Prior to that time, surgery was a self taught discipline among US physicians. He also invented and pioneered the use of rubber gloves. Unfortunately, much to his own regret, he began to use cocaine himself and became highly addicted to it. At that time, there was no stigma attached to the recreational use of cocaine, and it gained a following among the elites of the day. Arthur Conan Doyle's Sherlock Holmes was supposed to be an addict, and Holmes kept Dr Watson around as a source for his drugs, as well as for the comic relief he provided.

It became fairly obvious fairly quickly that while the anesthetic characteristics of cocaine were desirable, the euphoria and subsequent addiction it produced was not! The turn of the century was a tremendous time of scientific progress, and the new discipline of organic chemistry enabled the synthesis of the first analog of cocaine in 1905. (An analog of a chemical molecule is one in which the original molecule is progressively modified to retain and enhance certain holistic characteristics of the original substance while ridding it of other unwanted characteristics.) The first synthetic local anesthetic was procaine, better remembered today by its trade name, "Novocain".

Novocain was not without its problems. It took a very long time to set (ie. to produce the desired anesthetic result), wore off too quickly and was not nearly as potent as cocaine. On top of that, it is classified as an ester. Esters have a very high potential to cause allergic reactions. It is estimated that about one third of all persons who received it developed at least minor allergic reactions to it. Faced with the legal and ethical difficulties associated with the use of cocaine as a local anesthetic, and with the inefficiencies and allergenicity associated with the use of procaine, it is not surprising that most dentists of the day worked without any local anesthetic at all. (Nitrous oxide gas was available during this period.) Today, procaine is not even available for dental procedures.

The first modern local anesthetic agent was lidocaine (trade name Xylocaine®). It was invented in the 1940s. Prior to its introduction, Nitrous oxide gas (plus alcohol in the form of whiskey) was the major source of pain relief during dental procedures. Lidocaine proved to be so successful that during the 1940s and 1950s the use of nitrous oxide gas as a primary anesthetic agent all but vanished. (Whiskey somehow survived, but it is no longer used on patients.) Today, nitrous oxide is used principally as an anti-anxiety palliative. Lidocaine is in a broad class of chemicals called amides, and unlike ester based anesthetics, amides are hypoallergenic. It sets quickly and when combined with a small amount of epinephrine (adrenalin), it produces profound anesthesia for several hours. Lidocaine is still the most widely used local anesthetic in America today.

Over the next thirty years, a number of other amide local anesthetics were invented, most not differing significantly from lidocaine. The major problem with lidocaine and its analogs is that they cause vasodilation, or the tendency of the local blood vessels to open wider increasing the blood flow in the area. This causes the anesthetic to be absorbed too quickly to take effect. Hence these anesthetics are always mixed with low concentrations of epinephrine which has the opposite effect (ie vasoconstriction) and closes the blood vessels down to keep the anesthesia in position long enough to produce long lasting numbness.

Mepivicaine (Carbocaine®) and prilocaine (Citanest®) have much less vasodilative qualities and hence can be used without the epinephrine vasoconstrictor. The advantage to this is that these anesthetics can be used more safely in patients who are taking medications which may interact negatively with the vasoconstrictor. These drugs include certain blood pressure medications (most notably non selective beta blockers) and tricyclic antidepressants (Elevil® and imipramine are two examples). Carpules that do not contain the vasoconstrictor also do not contain a preservative. This eliminates a possible source of allergic reaction.

How nerves conduct an impulse



The image to the right is a fairly accurate representation of a nerve bundle. (For a detailed explanation of this diagram as well as nerve anatomy and physiology, see my page on Understanding Pain.) If you think of a nerve bundle as an electrical cable, the blue axons represent the "wires" that carry the impulse from the tooth to the ganglion at the other end. The rest of the tissue surrounding the axons represent the "insulation" which separates the various wires in the cable from each other. At this point, the analogy breaks down because, while the insulation in an electrical cable is a passive material that serves only to separate the wires from each other to prevent short circuits, the insulation in a nerve bundle is an active participant in the conduction of the impulse.

The connective tissue that is associated with each neuron is composed of a special material called myelin which is itself made up of the cell bodies of specialized cells called Schwann cells.

The myelin sheath is almost continuous along the entire axon. There are, however tiny breaks in the continuity of the myelin sheath between each succeeding Schwann cell. These breaks are called "nodes of Ranvier". These nodes are quite important in the conduction of an impulse along a nerve axon on its way to the cell body in the ganglion, mostly because their presence along the way speeds the impulse quite a bit.

How a nerve fiber transmits an impulse

Nerves are NOT like electrical wires with electrons traveling their length to transfer information from one end to the other. They are actually complex electro-chemical structures which utilize the electrical potential difference between the fluid inside of the axon, and the fluid that surrounds the axon. The fluid inside the axon (called cytoplasm) contains a high concentration of potassium ions, while the fluid outside contains a high concentration of sodium ions. There is no real difference in electrical potential between a potassium ion and a sodium ion, however, the fact that they exist in different concentrations on either side of the cell membrane sets up an electrochemical pressure gradient between the two. Sodium ions want to flow into the nerve cytoplasm, while the potassium ions want to flow out, but both are prevented from doing so by the presence of the nerve cell membrane.

When a nerve is stimulated, this sets up a chain reaction in which sodium ions begin to penetrate through the nerve cell membrane and flow into the axon, while potassium ions begin to flow out. This activity happens at the nodes of Ranvier. This process is called depolarization of the nerve membrane. The imbalance in the chemical makeup of the extracellular fluid then causes an imbalance in the concentration of sodium ions at the adjacent node which stimulates an identical depolarization at this node as well. This process proceeds from node to node until the impulse reaches the cell body of the nerve in the ganglion where it stimulates a similar cascade in a network of other neurons which make contact with it.

You might think that once all the potassium and sodium ions have exchanged places, the nerve would no longer be able to conduct impulses. The nerve, however, is a living entity and can regenerate the original concentrations of ions using energy from the food you eat in almost the same way that muscle cells use that same energy to cause muscle movement. It does this using proteins embedded in the cell membrane which act as "ion pumps".
http://www.doctorspiller.com/local_anesthetics.htm