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Lung Autopsies of COVID-19 Patients Reveal How Virus Spreads and Damages Tissue, Treatment Clues


Manwon Lender
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SARS-CoV-2 prevents lung tissue repair, and regeneration:

Although the study was small—lung samples from 18 cases and plasma samples from six of those cases—the scientists say their data revealed trends that could help develop new COVID-19 therapeutics and fine-tune when to use existing therapeutics at different stages of disease progression. The findings include details about how SARS-CoV-2, the virus that causes COVID-19, spreads in the lungs, manipulates the immune system, causes widespread thrombosis that does not resolve, and targets signaling pathways that promote lung failure, fibrosis and impair tissue repair. The researchers say the data are particularly relevant to caring for COVID-19 patients who are elderly, obese, or have diabetes—all considered high-risk populations for severe cases. Study samples were from patients who had at least one high-risk condition.

Lung Autopsies of COVID-19 Patients Reveal How Virus Spreads and Damages Tissue, Treatment Clues (scitechdaily.com)

Lung epithelial and endothelial damage, loss of tissue repair, inhibition of fibrinolysis, and cellular senescence in fatal COVID-19:  Below is the Peer Reviewed Journal for this article: Lung epithelial and endothelial damage, loss of tissue repair, inhibition of fibrinolysis, and cellular senescence in fatal COVID-19 (science.org)

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